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The increased glycolytic flux myocellular influence the reduction of fatty acid entry into mitochondria long chain. This assumption is based on the fact that indicate a lack of reduction in fatty acid oxidation medium chain during the second experimental condition, whose fatty acid oxidation would not be limited by Optimal Stack transport into mitochondria. One could speculate that an adequate supply of carbohydrates (with the consequent increase in its catabolism) inhibits the transport of long-chain fatty acids into mitochondria, thus controlling the metabolic mixture during exercise (Wolfe, RR, 1998). This proposal, however, is directly opposed to a classical hypothesis which states that the availability of fatty acids, mediated lipolysis, inhibits the metabolism of carbohydrates, as described by the cycle of the glucose-fatty acid (Garland, PB, Randle, PJ, 1964).


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